Patients are typically started on anticoagulation depending on their thrombotic and bleeding risk.Ītrial flutter is another common type of supraventricular tachyarrhythmia that is usually caused by a single macroreentrant rhythm within the atria. Rhythm control strategies include synchronized electrical cardioversion, the use of pharmacological antiarrhythmics (e.g., flecainide, propafenone, or amiodarone), and ablation of the arrhythmogenic tissue. Rate-control therapy typically involves the use of beta blockers or nondihydropyridine calcium channel blockers. In stable patients, treatment involves the correction of modifiable risk factors, rate or rhythm control strategies, and anticoagulation. Immediate synchronized cardioversion is required in hemodynamically unstable patients. Echocardiography is used to rule out structural heart disease and to evaluate for any atrial thrombi. Diagnosis is confirmed with ECG showing absent P waves with irregular QRS intervals. Ineffective atrial emptying as a result of Afib can lead to stagnation of blood and clot formation in the atria, which in turn increases the risk of stroke and other thromboembolic complications. Physical examination typically reveals an irregularly irregular pulse. When symptoms do occur, they usually include palpitations, lightheadedness, and shortness of breath.

Individuals with Afib are typically asymptomatic. While the exact mechanisms of Afib are poorly understood, associations with a number of cardiac (e.g., valvular heart disease, coronary artery disease) and noncardiac (e.g., hyperthyroidism, electrolyte imbalances) risk factors have been established. Atrial fibrillation (Afib) is a common type of supraventricular tachyarrhythmia characterized by uncoordinated atrial activation that results in an irregular ventricular response.

Difference between a flutter and afib